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The endpoint outcome, rather than any safety signal, is what redirected the program

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(2001, Int J Obes) showed that chronic treatment with AOD-9604 restored repressed beta-3 AR mRNA levels in obese mice toward lean-mouse values, characterizing a chronic-treatment mechanism that complements the fragment's acute lipolytic action

Immunofluorescence staining targeted neurogenic markers including nestin, GAP43, -tubulin III, and doublecortin

Therefore, an improved ADHD treatment may strengthen dopaminergic neurotransmission without directly blocking DAT